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Shibata, T., et al.,Cancer related mutations in NRF2 impair its recognition by Keap1-Cul3 E3 ligase and promote malignancy

Fasting is required for CR-induced improvements in tau hyperphosphorylation and neuroinflammation, but not for CR-induced improvements in A plaque deposition To investigate the role of fasting in the ability of CR to attenuate AD pathology, we evaluated several pathological hallmarks of AD, including phosphorylation of tau, amyloid beta (A) plaque deposition, and gliosis

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Anti-POMC and anti-c-Fos antibodies were used (Supplementary Data 1)

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To determine dose level and dose frequency of central administration of mGIPR-Ab, pharmacokinetic profile of ( a ) brain and ( b ) plasma exposure was assessed at 4, 24, 72, 120, and 168 h following a single ICV injection of 15 g/L/mouse dose of mGIPR-Ab