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However, it was not clear how ASA exerts its multiple effects in oxidative stress conditions when cellular GSH pool is altered (depleted or enhanced)

Previous research has shown that immuno-metabolic dysregulations map more consistently to symptoms of the atypical spectrum characterized by altered energy intake/output (in particular the reversed neurovegetative symptoms of hyperphagia, hypersomnia with leaden paralysis and fatigue) [2] and symptoms of anhedonia [33]

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or (3) placebo for six months

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