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Increased levels of lipid peroxidation, protein oxidation, and DNA damage have been repeatedly found in ASD patients, indicating generalized oxidative damage and mitochondrial impairment

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[2] BPC-157 and Nitric Oxide Signaling: The Core Cardiovascular Mechanism The most reproducible cardiovascular effect of BPC-157 is upregulation of the nitric oxide (NO) pathway, specifically through endothelial nitric oxide synthase (eNOS)

The relationship between Na 2 S 2 concentration and inhibition time (left axis) allows quantification of eliminated radicals (right axis), using a calibration curve (right panel)

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