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In the Glp1r VSM/ mouse, the transgene is inserted on the Y chromosome, precluding the study of female mice in this model

This mechanism raises the critical discontinuation question - does stopping GHK-Cu immediately halt enhanced collagen production returning synthesis rates to pre-treatment baseline, or does the improved dermal environment created during treatment continue supporting elevated collagen production through positive feedback mechanisms where better matrix structure itself promotes ongoing synthesis even without continuous peptide stimulation

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It may help to reduce scarring and improve the appearance of the skin after injuries or surgeries

This is a tactful suggestion, not a prescription

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